Solar Storms for Lightworkers

30 07 2013

Due to recent posts online with regard to (remote viewing) “End Times” scenarios related to solar flares, here’s the “101” on SOLAR STORMS.

There are three types: (1) Solar Flares, (2) Solar Proton Events (SPE), and (3) Coronal Mass Ejections (CME).

BTW, before delving into these three types, my suggested solution (as Lightworkers) is to focus meditation on NORMAL solar activities and strengthened magnetic field to shield the planet–even better would be to step up meditation to focus on the geometrical grid known as the “flower of life” to protect the Earth at all times.

(1) Type 1: Solar Flares. Well, the good news is that we won’t burn to a crisp in a solar flare (C- or M-class), b/c the electromagnetic radiation (in the form of X-rays, gamma rays and extreme UV rays) cannot penetrate the Earth’s atmosphere (or geomagnetic shield) but can only reach the polar regions where it loses energy despite arriving from the sun within a couple hours. It can, however, interfere with our daily lives by interrupting communications satellites as well as cause shortwave radio blackouts. That means no use of communications technologies that rely on satellites. X-class solar flares are a different story and the worst of its kind. Either way, none of these rays are visible to the naked eye. We wouldn’t even see them coming.

(2) Type 2: Solar Proton Events or SPE are “high-energy solar cosmic rays (protons and ions)” that get to Earth in 15 min to a couple hours. This type is not nearly as friendly as type 1 especially as it pertains to the impact on human physiology. Sure, it has the same effects on communications technology such as “satellite disorientation and shortwave radio fades” in addition to “disruption in polar regions, and ozone layer depletion”, but it also affects the human anatomy causing “cardiac arrest, dementia and [high radiation] cancer”. “If the particles have energies greater than 500 MeV, the deadly cosmic ray shower can penetrate to the planet’s surface” (right through the Earth’s atmospheric shield)—96% of 500 MeV SPEs are followed by type 3, below.

(3) Coronal Mass Ejections (CME) comprise “vast clouds of seething gas, charged PLASMA of low to medium energy particles with an imbedded magnetic field, blasted into interplanetary space from the Sun. This causes a worldwide disturbance of Earth’s magnetic field called a geomagnetic storm that produces a temporary disturbance of the Earth’s magnetosphere, etc… A CME can produce the following effects: …radio propagation anomalies, compass alignment errors, electrical power blackouts, oil and gas pipeline corrosion, communication landline & equipment damage, electrical shock hazard, electrical fires, heart attacks, strokes, and workplace & traffic accidents.” Depending on the direction of the geomagnetic storm and the orientation of the Earth’s magnetic field, a storm headed southward would yield the most severe effects on the planet.

KILL SHOT

These solar flare concerns were sparked by remote viewing predictions claiming Earth is entering its End Times, per Ed Dames, “Kill Shot”:

Here’s version of the longer interview on Coast to Coast:

Note, while some predictions have proven accurate, not all black budget military remote viewing scenarios have come true (including those of Ed Dames).

MASS DREAMS OF THE FUTURE | HOLOGRAPHIC UNIVERSE

Interestingly, in a study done over 30 years ago, Helen Wambach applied “hypnotic progression” or hypnosis to study the future. In a sampling of 2,500 participants, nearly all saw major depopulation and many were no longer in physical form in the future.  The results yielded roughly four main post-2012 End Times scenarios depending on the belief systems (or FREQUENCY/VIBRATION) of each person as (1) the hypnotist AND (2) those whose future were being reported.

Wambach later died prior to finishing her study, but her colleague Chet B. Snow published the results  in the book “Mass Dreams of the Future”.  They are as follows:

FOUR SCENARIOS:

(1)  GROUP A: NATURAL DISASTERS – human populations were moved to space stations living sterile and unhappy lives, eating synthetic foods and wearing space suits.

(2) GROUP B: NEW AGERS – experienced ascension or The Golden Age of Love/Photon Belt, Peace, and Awakening/Self-Realization as Creator-Creation and were living happier and more natural lives in harmony with each other.

(3) GROUP C: HI-TECH URBANITES – saw a bleak mechanical future in which people lived in subterranean cities and cities under bio domes.

(4) GROUP D – POST DISASTER SURVIVORS – (either nuclear or natural cataclysmic) who lived in urban ruins from caves to isolated farms and obtained much of their food from hunting.

RAISING FREQUENCY

One’s frequency always determines his/her future “timeline” (among its infinite probabilities). So long as each of us continues to be Love, meditate Love, give Love, and spread Love as Emissaries of the Divine Light and according to Godfrye’s “St. Germain, Unveiled Mysteries”: “Love the Light, Serve the Light, Live in the Light, Bless the Light, be Protected, Illumined, Sustained and Supplied by the Light”, timeline #2 is the only reality one would experience.

GALACTIC ASSISTANCE

In a similar look into the future, there might have been another category in which nothing happened at all, at least not to the conscious awareness of the majority of Earth’s inhabitants (with the exception of those aware and awake who consciously call on the assistance of the higher fields of Light via meditation, prayer, channeling, etc).  This timeline might be accredited to the dedicated and scientifically advanced assistance of our galactic friends who are already known to have been of considerable service to humanity in recent years and decades (e.g. blasting comet Elenin, blasting the larger meteor that was reduced to the smaller Russian asteroid, redirecting the mini-solar system of Nibiru, disabling nuclear weapons worldwide for decades, diffusing and/or clearing radiation disasters such as Fukushima, subduing undersea Reptilian military bases, sedating super volcanoes e.g. Yellowstone and the Canary Islands, etc.) We are certainly not alone, at least not without assistance said to come from the united forces of our galactic family of Light such as the Pleiadians, Sirians, Andromedans, Arcturans, Krulians, Alpha Centaurians, and many more.

by Mae McCaw

Sources:

  1. http://www.breadandbutterscience.com/SSTA.pdf
  2. Mass Dreams of the Future by Chet B. Snow
  3. Unveiled Mysteries (Saint Germain Series, Vol. 1) by Godfré Ray King

Suspicious0bservers (Youtube Channel) does a great job of covering solar and planetary weather activities with its “4-minute” daily report.





Chemtrails, Geoengineering and Methane Mass Expulsion

27 07 2013

The Most Important Topic For 2013

Geoengineering: The Real Climate Threat

Fox Primetime News Report on Chemtrails

USAF whistle blower testimony

“Chemtrails and Barium Toxicity” by Dr. McCain:

Toxicity Profiles Toxicity Summary for BARIUM NOTE – Although the toxicity values presented in these toxicity profiles were correct at the time they were produced, these values are subject to change. Users should always refer to the Toxicity Value Database for the current toxicity values.

EXECUTIVE SUMMARY
1. INTRODUCTION
2. METABOLISM AND DISPOSITION
2.1 ABSORPTION

DISTRIBUTION
2.3 METABOLISM
2.4 EXCRETION
3. NONCARCINOGENIC HEALTH EFFECTS
3.1 ORAL EXPOSURES

INHALATION EXPOSURES
3.3 OTHER ROUTES OF EXPOSURE
3.4 TARGET ORGANS/CRITICAL EFFECTS
4. CARCINOGENICITY
4.1 ORAL EXPOSURES

INHALATION EXPOSURES
4.3 OTHER ROUTES OF EXPOSURE
4.4 EPA WEIGHT-OF-EVIDENCE
4.5 CARCINOGENICITY SLOPE FACTORS

REFERENCES

Prepared by A. A. Francis, M.S., D.A.B.T., and Carol S. Forsyth, Ph.D., Chemical Hazard Evaluation Group in the Biomedical and Environmental Information Analysis Section, Health Sciences Research Division, *.

Prepared for OAK RIDGE RESERVATION ENVIRONMENTAL RESTORATION PROGRAM

*Managed by Martin Marietta Energy Systems, Inc., for the U.S. Department of Energy under Contract No. DE-AC05-84OR21400.

EXECUTIVE SUMMARY

The soluble salts of barium, an alkaline earth metal, are toxic in mammalian systems. They are absorbed rapidly from the gastrointestinal tract and are deposited in the muscles, lungs, and bone. Barium is excreted primarily in the feces.

At low doses, barium acts as a muscle stimulant and at higher doses affects the nervous system eventually leading to paralysis. Acute and subchronic oral doses of barium cause vomiting and diarrhea, followed by decreased heart rate and elevated blood pressure. Higher doses result in cardiac irregularities, weakness, tremors, anxiety, and dyspnea. A drop in serum potassium may account for some of the symptoms. Death can occur from cardiac and respiratory failure. Acute doses around 0.8 grams can be fatal to humans.

Subchronic and chronic oral or inhalation exposure primarily affects the cardiovascular system resulting in elevated blood pressure. A lowest-observed-adverse-effect level (LOAEL) of 0.51 mg barium/kg/day based on increased blood pressure was observed in chronic oral rat studies (Perry et al. 1983), whereas human studies identified a no-observed-adverse-effect level (NOAEL) of 0.21 mg barium/kg/day (Wones et al. 1990, Brenniman and Levy 1984). The human data were used by the EPA to calculate a chronic and subchronic oral reference dose (RfD) of 0.07 mg/kg/day (EPA 1995a,b). In the Wones et al. study, human volunteers were given barium up to 10 mg/L in drinking water for 10 weeks. No clinically significant effects were observed. An epidemiological study was conducted by Brenniman and Levy in which human populations ingesting 2 to 10 mg/L of barium in drinking water were compared to a population ingesting 0 to 0.2 mg/L. No significant individual differences were seen; however, a significantly higher mortality rate from all combined cardiovascular diseases was observed with the higher barium level in the 65+ age group. The average barium concentration was 7.3 mg/L, which corresponds to a dose of 0.20 mg/kg/day. Confidence in the oral RfD is rated medium by the EPA.

Subchronic and chronic inhalation exposure of human populations to barium-containing dust can result in a benign pneumoconiosis called “baritosis.” This condition is often accompanied by an elevated blood pressure but does not result in a change in pulmonary function. Exposure to an air concentration of 5.2 mg barium carbonate/m3 for 4 hours/day for 6 months has been reported to result in elevated blood pressure and decreased body weight gain in rats (Tarasenko et al. 1977). Reproduction and developmental effects were also observed. Increased fetal mortality was seen after untreated females were mated with males exposed to 5.2 mg/m3 of barium carbonate. Similar results were obtained with female rats treated with 13.4 mg barium carbonate/m3. The NOAEL for developmental effects was 1.15 mg/m3 (equivalent to 0.8 mg barium/m3). An inhalation reference concentration (RfC) of 0.005 mg/m3 for subchronic and 0.0005 mg/m3 for chronic exposure was calculated by the EPA based on the NOAEL for developmental effects (EPA 1995a). These effects have not been substantiated in humans or other animal systems.

Barium has not been evaluated by the EPA for evidence of human carcinogenic potential (EPA 1995b).

1. INTRODUCTION

Barium (CAS registry number 7440-39-3) is a divalent alkaline-earth metal found only in combination with other elements in nature. The most important of these combinations are the peroxide, chloride, sulfate, carbonate, nitrate, and chlorate. The pure metal oxidizes readily and reacts with water emitting hydrogen; it is chemically similar to calcium (Weast et al. 1987). The most likely source of barium in the atmosphere is from industrial emissions. Since it is usually present as a particulate form, it can be removed from the atmosphere by wet precipitation and deposition. Due to the element’s tendency to form salts with limited solubility in soil and water, it is expected to have a residence time of hundreds of years and is not expected to be very mobile. Acidic conditions, however, will increase the solubility of some barium compounds facilitating their movement from the soil to the groundwater (EPA 1984). Trace amounts of barium were found in more than 99% of the surface waters and finished drinking water samples (average values of 43 g/L, and 28.6 g/L, respectively) across the United States (National Academy of Sciences 1977).

2. METABOLISM AND DISPOSITION

2.1 ABSORPTION

The soluble forms of barium salts are rapidly absorbed into the blood from the intestinal tract. The rates of absorption of a number of barium salts have been measured in rats following oral exposure to small quantities (30 mg/kg body weight). The relative absorption rates were found to be: barium chloride barium sulfate barium carbonate. Large doses of barium sulfate do not increase the uptake of this salt because of its low solubility (McCauley and Washington 1983, EPA 1984).

Systemic toxic effects have been observed following both oral and inhalation exposure. No absorption kinetics are available following inhalation exposure, although it is obvious that absorption does occur (EPA 1984).

2.2 DISTRIBUTION

Barium absorbed into the bloodstream disappears in about 24 hours; however, it is deposited in the muscles, lungs, and bone. Very little is stored in the kidneys, liver, spleen, brain, heart, or hair. It remains in the muscles about 30 hours after which the concentration decreases slowly. The deposition of barium into bone is similar to calcium but occurs at a faster rate (Beliles 1994). The half life of barium in bone is estimated to be about 50 days (Machata 1988).

2.3 METABOLISM

About 54% of the barium dose is protein bound. Barium is known to activate the secretion of catecholamines from the adrenal medulla without prior calcium deprivation. It may displace calcium from the cell membranes, thereby increasing permeability and providing stimulation to muscles. Eventual paralysis of the central nervous system can occur (Beliles 1994).

2.4 EXCRETION

A tracer study in rats using 140Ba demonstrated that 7% and 20% of the barium dose was excreted in 24 hours in the urine and feces, respectively. In contrast, calcium is primarily excreted in the urine. The clearance of barium is enhanced with saline infusion (Beliles 1994). Following intravenous injection of barium into six healthy men, excretion was mainly fecal with the total relative fecal:urinary clearance for 14 days ranging from 6 to 15 (Newton et al. 1991).

3. NONCARCINOGENIC HEALTH EFFECTS

3.1 ORAL EXPOSURES

3.1.1 Acute Toxicity

3.1.1.1 Human

A number of accidental barium poisonings have occurred following the ingestion of barium salts. The estimated fatal dose of barium carbonate, a rodenticide, is about 5 grams for a 70 kg human (Arena 1979). The LD50 for barium chloride is estimated at about 1 gram for a 70 kg human (Machata 1988), and the LDLo (lowest published lethal dose) is reported to be about 0.8 grams (Lewis and Sweet 1984). The acute symptoms include excess salivation, vomiting, diarrhea, increased blood pressure, muscular tremors, weakness, paresis, anxiety, dyspnea, and cardiac irregularities. A severe loss of potassium can account for some of the symptoms. Convulsions and death from cardiac and respiratory failure can occur. Magnesium and sodium sulfate are antidotal if taken soon after ingestion since either salt will result in the formation of insoluble barium sulfate and prevent further absorption. Survival for more than 24 hours is usually followed by complete recovery (Arena 1979).

Complications occurred in a woman following a barium swallow investigation for severe dysphagia. Direct aspiration of a large amount of barium into the right main bronchus resulted in tachycardia, tachypnoea, fever, and an oxygen saturation of 82%; two weeks later the woman still had a moist cough with widespread rales but continued to recover (Penington 1993).

A family was accidentally poisoned with barium from eating their evening meal. The mother had fried fish breaded with a flour-like substance that turned out to be rat poison containing barium carbonate. All seven family members, aged 2 to 48 years, developed nausea, vomiting, diarrhea, and crampy abdominal pain within minutes of consuming the meal; the parents also developed ventricular tachycardia, flaccid paralysis of the extremities, shortness of breath (mother), and respiratory failure (father). Patients were treated symptomatically and all fully recovered (Johnson and VanTassell 1991).

3.1.1.2 Animal

Similar acute symptoms occur in animals; however, higher doses are usually involved. The LD50 for rats is listed as 630 mg/kg for barium carbonate, 118 mg/kg for barium chloride, and 921 mg/kg for barium acetate (Lewis and Sweet 1984).

3.1.2 Subchronic Toxicity

3.1.2.1 Human

An experiment testing the subchronic toxicity of barium chloride on human volunteers was conducted by Wones et al. (1990). The diets of 11 male subjects were controlled. They were given 1.5 L/day of distilled and charcoal-filtered drinking water that contained 0 mg/L barium for weeks 1 and 2, 5 mg/L for weeks 3 to 6, and 10 mg/L for weeks 7 to 10. No clinically significant effects were observed in blood pressures, serum chemistry, urinalysis, or electrocardiograms. The 10 mg/L (0.21 mg/kg/day) dose was identified as a NOAEL.

3.1.2.2 Animal

Groups of 30 male and 30 female Charles River rats were exposed to barium chloride at 0, 10, 50, or 250 ppm in drinking water for 90 days (Tardiff et al. 1980). The highest average dose in this study was calculated to be 45.7 mg/kg/day for female rats. No significant clinical signs of toxicity were observed. Blood pressure was not measured.

McCauley et al. (1985) conducted drinking water studies in which six male Sprague-Dawley rats/group were given water containing 0, 10, 100, or 250 mg/L barium for 36 weeks, or 1, 10, 100, or 1000 mg/L barium for 16 weeks. Female rats were given 0 or 250 mg/L for 46 weeks. Animals receiving the 1000 mg/L dose developed ultrastructural changes in the kidney glomeruli. No other effects were reported.

Tardiff et al. (1980) exposed groups of 30 male and 30 female Charles River rats to 0, 10, 50, or 250 ppm barium (given as barium chloride) in drinking water for 90 days. A slight reduction in adrenal weights was seen in female rats with the 250 ppm (45.7 mg/kg/day) dose at 13 weeks, and no other adverse effects were observed in male rats with the 50 ppm (8.1 mg/kg/day) and the 250 ppm (38.1 mg/kg/day) doses at 8 weeks. No clear dose effect or dose duration effect was seen with the adrenal weight decrease; therefore, the clinical significance is uncertain.

3.1.3 Chronic Toxicity

3.1.3.1 Human

An epidemiology study conducted by Brenniman and Levy (1984) compared a human population ingesting barium levels of 2 to 10 mg/L in their drinking water to a population ingesting 0 to 0.2 mg/L. Although significantly higher mortality rates from all cardiovascular diseases were observed with the higher barium level in the 65 and over age group, there were no significant individual differences in blood pressures, strokes, or heart and renal diseases within the two groups. The average barium concentration for the mortality study was 7.3 mg/L, which corresponds to a dose of 0.20 mg/kg/day assuming drinking water consumption of 2 L/day for a 70 kg human.

3.1.3.2 Animal

A series of experiments were performed in which groups of 52 male and female Long-Evans rats and 42 male and female Swiss mice were exposed to 5 mg barium/L (given as barium acetate) in drinking water for their lifetime (Schroeder and Mitchener 1975a,b). The barium doses were about 0.25 and 0.825 mg/kg/day for rats and mice, respectively. No adverse clinical effects were observed; however, blood pressure was not measured. A slight but significant reduction in longevity of treated male mice was noted when measured as the mean age at death of the last surviving 10% of animals. The overall average life span of the group, however, was about the same as the control group (EPA 1984, 1989).

Perry et al. (1983) exposed 12 to 13 female weanling rats/group to 0, 1, 10, or 100 ppm barium (given as barium chloride) for up to 16 months. Average doses were calculated to be 0, 0.051, 0.51, and 5.1 mg/kg/day (EPA 1985). A clinically significant increase in average blood pressure was observed in the highest dose group; a slight but statistically significant increase was seen in the 10 ppm (0.51 mg/kg/day) dose group. The controlled diet, which restricted the intake of trace metals, calcium, and potassium, may have contributed to the effect.

3.1.4 Developmental and Reproductive Toxicity

Information on developmental and reproductive toxicity in humans or animals following oral exposure was unavailable.

3.1.5 Reference Dose

3.1.5.1 Subchronic

ORAL RfDs: 0.07 mg/kg/day (EPA 1995a)

UNCERTAINTY FACTOR: 3

NOAEL: 0.21 mg/kg/day

PRINCIPAL STUDIES: The same studies and comments apply to both the subchronic and chronic RfD derivations. See Sect. 3.1.5.2.

3.1.5.2 Chronic

ORAL RfDc: 0.07 mg/kg/day (EPA 1995b)

UNCERTAINTY FACTOR: 3

MODIFYING FACTOR: 1

NOAEL: 0.21 mg/kg/day

CONFIDENCE:

Study: Medium

Data Base: Medium

RfD: Medium

VERIFICATION DATE: 06/21/90

PRINCIPAL STUDIES: Wones et al. (1990); Brenniman and Levy (1984).

COMMENTS: The RfD values are based on a weight-of-evidence approach using subchronic to chronic human drinking water studies. The uncertainty factor accounts for protecting sensitive individuals and is reduced from the usual factor of 10 because the selected studies examined the population judged most at risk.

3.2 INHALATION EXPOSURES

3.2.1 Acute Toxicity

3.2.1.1 Human

Barium carbonate dust has been reported to be a bronchial irritant. Barium oxide dust is considered a dermal and nasal irritant (Beliles 1994). The effect of barium dusts on welders was investigated under simulated working conditions over a one-week time period (Zschiesche et al. 1992). Barium fume concentrations were 4.4 and 2.0 mg/m3 during welding with stick electrodes and flux cored wires, respectively. No adverse health effects on the welders were attributable to barium exposure, but there was a slight decrease in plasma potassium levels at the end of the work shift.

3.2.1.2 Animal

Information on the acute inhalation toxicity of barium in animalswas not available.

3.2.2 Subchronic Toxicity

3.2.2.1 Human

Industrial workers exposed to barium dust, usually in the form of barium sulfate or carbonate, often develop a benign pneumoconiosis referred to as “baritosis.” Because of the radiopacity of barium compounds, this condition can be specifically diagnosed radiologically. After removal from the sources of exposure, baritosis is reversible in most cases. Baritosis results in a significantly higher incidence of hypertension, but no changes are usually seen in pulmonary function (Stokinger 1981, EPA 1995b).

3.2.2.2 Animal

Male rats were exposed to 1.15 and 5.2 mg/m3 of barium carbonate dust for 4 hours/day for 6 months. The high dose animals developed increased arterial pressure; decreased body weight gain; decreased blood levels of hemoglobin, sugar, protein, cholinesterase and thrombocytes; increased blood levels of leukocytes, phosphorous and alkaline phosphatase; increased urine calcium; and perivascular and peribronchial sclerosis in the lungs. (EPA 1984, Tarasenko et al. 1977).

3.2.3 Chronic Toxicity

3.2.3.1 Human

Baritosis and bronchial irritation have been reported in workers chronically exposed to barium containing dust (Beliles 1994).

3.2.3.2 Animal

Information on the chronic inhalation toxicity of barium in animals was not available.

3.2.4 Developmental and Reproductive Toxicity

Tarasenko et al. (1977) performed a series of experiments in rats designed to test for possible reproductive and developmental effects. Increased fetal mortality was observed following the mating of males exposed to barium carbonate (5.2 mg/m3 air) with untreated females. Decreased sperm motility was observed in males treated with 22.6 mg/m3. The mating of females exposed to 13.4 mg/m3 for 4 months also resulted in increased fetal mortality and a general under development of the newborn pups. Ovarian follicle atresia was seen in female rats exposed to 3.1 mg/m3. No significant adverse effects were noted with the 1.15 mg/m3 concentration (EPA 1984).

3.2.5 Reference Concentration/Dose

3.2.5.1 Subchronic

INHALATION RfCs: 0.005 mg/m3; 0.001 mg/kg/day (EPA 1995a)

UNCERTAINTY FACTOR: 100

NOEL: 0.8 mg Ba/m3 given 4 hr/day (EPA 1995a)

PRINCIPAL STUDY: The same study and comments apply to the subchronicand chronic RfC. The study is described in Sect. 3.2.4.

3.2.5.2 Chronic

INHALATION RfCc: 0.0005 mg/m3; 0.0001 mg/kg/day (EPA 1995a)

UNCERTAINTY FACTOR: 1000

NOEL: 0.8 mg Ba/m3 given 4 hr/day (EPA 1995a)

PRINCIPAL STUDY: Tarasenko et al. 1977

COMMENTS: The dose of 1.15 mg BaCO3/m3 was given as the NOEL in the principal study, which is equivalent to 0.8 mg barium/m3 used as the basis for the RfC calculations. An inhalation risk assessment for barium is under review by an EPA work group (EPA, 1995b).

3.3 OTHER ROUTES OF EXPOSURE

3.3.1 Acute Toxicity

3.3.1.1 Humans

Barium oxide dust is considered to be a dermal and nasal irritant(Beliles 1994).

3.3.1.2 Animal

A number of experiments have used intravenous and subcutaneous injections to measure lethal levels of soluble barium compounds. LD50 values for barium chloride, nitrate, and acetate were determined in two strains of mice by intravenous injection (Syed and Hosain 1972). The affected animals either died within one hour or survived the treatment. The LD50 values obtained were 8.12, 8.49, and 11.32 mg barium/kg for the chloride, nitrate, and acetate, respectively, in Swiss-Webster mice, and 19.20, 20.10, and 23.31 mg barium/kg for the chloride, nitrate, and acetate, respectively, in ICR mice. Although the relative toxicity of the barium salts remained the same, there was an unexplained two-fold difference in the LD50 values between the two mice strains.

The LDLo has been determined by subcutaneous injection in mice to be 10 mg/kg for the barium nitrate and chloride salts. The LDLo in rabbits was 55 mg/kg for the chloride and 96 mg/kg for the acetate salts. The LDLo values vary widely with the route and test animal. For example, with barium chloride the LDLo value for oral administration to rabbits is 170 mg/kg, whereas the value for subcutaneous injection is 55 mg/kg. Subcutaneous injection in mice results in a value of 10 mg/kg, which is higher than the LD50 value for intravenous injection in the Swiss-Webster strain (Lewis and Sweet 1984).

3.3.2 Subchronic Toxicity

Information on the subchronic toxicity of barium in humans andanimals was not available.

3.3.3 Chronic Toxicity

Information on the chronic toxicity of barium in humans and animalswas not available.

3.3.4 Developmental and Reproductive Toxicity

Information on the developmental and reproductive toxicity of bariumin humans and animals was not available.

3.4 TARGET ORGANS/CRITICAL EFFECTS

3.4.1 Oral Exposures

3.4.1.1 Primary Target Organs

1. Cardiovascular system: Subchronic to chronic symptoms include increased blood pressure and increased incidence of cardiovascular disease in humans. An acute overdose can result in cardiac irregularities. Convulsions and death from cardiac and respiratory failure can occur.

2. Nervous system: Acute to subchronic symptoms include weakness, tremors, anxiety, and dyspnea. An acute overdose can result in convulsions and death from cardiac and respiratory failure.

3.4.1.2 Other targets  Gastrointestinal system: Acute to subchronic symptoms include excess salivation, vomiting, and diarrhea in humans.

3.4.2 Inhalation Exposures

3.4.2.1 Primary target(s)

1. Cardiovascular system: Symptoms include increased blood pressure in humans.

2. Reproduction and development: Subchronic exposure of rats resulted in decreased sperm motility and ovarian follicle atresia. Increased fetal mortality and underdevelopment of newborn pups were also reported.

3.4.2.2 Other target(s)  Lungs: Subchronic to chronic exposure in humans results in a pneumoconiosis known as “baritosis” that usually does not adversely affect pulmonary function.

4. CARCINOGENICITY

4.1 ORAL EXPOSURES

4.1.1 Human

Information on the carcinogenicity of barium in humans was notavailable.

4.1.2 Animal

No significant differences in tumor incidence were found in either rats or mice in the lifetime exposure experiments of Schroeder and Mitchener (1975a,b), as described in Sect. 3.1.3.2.

4.2 INHALATION EXPOSURES
Information on the carcinogenicity of barium in humans and animalswas not available.

4.3 OTHER ROUTES OF EXPOSURE
Information on the carcinogenicity of barium in humans and animalswas not available.

4.4 EPA WEIGHT-OF-EVIDENCE
Barium has not been evaluated by the EPA for evidence of humancarcinogenic potential (EPA 1995b).

4.5 CARCINOGENICITY SLOPE FACTORS

Data are insufficient to calculate a slope factor for barium.

5. REFERENCES

  1. Arena, J. M. 1979. Poisoning – Toxicology – Symptoms – Treatments.Charles C. Thomas, Publisher, Springfield, Ill. pp. 173-179.
  2. Beliles, R. P. 1994. The Metals. In: Patty’s Industrial Hygiene and Toxicology, 4th ed., G.D. Clayton and F. E. Clayton, eds. John Wiley& Sons, New York. pp. 1925-1929.
  3. Brenniman, G. R. and P. S. Levy. 1984. High barium levels in public drinking water and its association with elevated blood pressure. In:Advances in Modern Toxicology IX, E. J. Calabrese, Ed. Princeton Scientific Publications, Princeton, NJ. pp. 231-249.
  4. EPA (United States Environmental Protection Agency). 1984. Health Effects Assessment for Barium. Prepared by the Office of Health and Environmental Assessment, Environmental Criteria and Assessment Office, Cincinnati, OH, for the Office of Emergency and Remedial Response, Washington, D.C.
  5. EPA. 1985. Drinking Water Health Effects Criteria Document on Barium. NTIS PB 86-118031. Prepared by the Office of Health and Environmental Assessment, Environmental Criteria And Assessment Office, Cincinnati, OH for the Office of Drinking Water, Washington, D.C.
  6. EPA. 1989. Reportable Quantity Document for Barium and Compounds.Prepared by the Office of Health and Environmental Assessment,Environmental Criteria and Assessment Office, Cincinnati, OH, for the Office of Solid Waste and Emergency Response, Washington, D.C.
  7. EPA. 1995a. Health Effects Assessment Summary Tables. Annual FY-95.Prepared by the Office of Health and Environmental Assessment,Environmental Criteria and Assessment Office, Cincinnati, OH, for the Office of Emergency and Remedial Response, Washington D.C.
  8. EPA. 1995b. Integrated Risk Information System (IRIS). Health Risk Assessment for Barium. On line. (Verification date 6/21/90.) Office of Health and Environmental Assessment, Environmental Criteria and Assessment Office, Cincinnati, OH. Retrieved 4/5/95.
  9. Johnson, C. H. and V. J. Van Tassell. 1991. Acute barium poisoning with respiratory failure and rhabdomyolysis. Ann. Emer. Med.20:1138-1142.
  10. Lewis, R. J. and D. V. Sweet, eds. 1984. Registry of Toxic Effects of Chemical Substances, Vol. 1. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control,National Institute for Occupational Safety and Health, Cincinnati, OH.
  11. Machata, G. 1988. Barium. In: Handbook on Toxicity of Inorganic Compounds, H. G. Seiler and H. Sigel, eds., Marcel Dekker, Inc. pp.97-101.
  12. McCauley, P. T. and I. S. Washington. 1983. Barium bio availability as the chloride, sulfate or carbonate salt in the rat. Drug Chem. Toxicol. 6(2):209-217.
  13. McCauley, P. T., B. H. Douglas, R. D. Laurie, and R. J. Bull. 1985.Investigations into the effect of drinking water barium on rats.Environ. Health Perspect. Vol. IX, E. J. Calabrese, ed. Princeton Scientific Publications, Princeton, NJ. pp.197-210.
  14. National Academy of Sciences. 1977. Drinking Water and Health. Safe Drinking Water Committee, Advisory Center on Toxicology, Assembly ofLife Sciences, National Research Council. Washington, D.C. pp211-212.
  15. Newton, D., G. E. Harrison, C. Kang, and A. J. Warner. 1991.Metabolism of injected barium in sex healthy men. Health Physics61:191-201.
  16. Penington, G. R. 1993. Severe complications following a “bariumswallow” investigation for dysphagia. Med. J. Aust. 159:764-765.
  17. Perry, H. M., S. J. Kopp, M. W. Erlanger, and E. F. Perry. 1983.Cardiovascular effects of chronic barium ingestion. In: TraceSubstances in Environmental Health, XVII, D. D. Hemphill, ed. Proc.Univ. Missouri’s 17th Ann. Conf. on Trace Substances inEnvironmental Health. University of Missouri Press, Columbia, MO. pp.155-164.
  18. Schroeder, H. and M. Mitchener. 1975a. Life-term studies in rats:Effects of aluminum, barium, beryllium and tungsten. J. Nutr.105:421-427.
  19. Schroeder, H. and M. Mitchener. 1975b. Life-term effects of mercury,methyl mercury and nine other trace metals on mice. J. Nutr. 105:452-458.
  20. Stokinger, H. E. 1981. The Metals. In: Patty’s Industrial Hygieneand Toxicology, 3rd ed., G.D. Clayton and F.E. Clayton, eds. JohnWiley & Sons, New York. pp. 1531-1537.
  21. Syed, I. B. and F. Hosain. 1972. Determination of LD50 of BariumChloride and Allied Agents. Toxicol. Appl. Pharm. 22:150-152.
  22. Tarasenko, M, O. Promin, and A. Silayev. 1977. Barium compounds asindustrial poisons (an experimental study). J. Hyg. Epidem.Microbiol. Immunol. 21:361-373.
  23. Tardiff, R. G., M. Robinson, and N. S. Ulmer. 1980. Subchronic oraltoxicity of barium chloride in rats. J. Environ. Pathol. Toxicol.4(5-6):267-276.
  24. Weast, R. C., J. A. Melvin, and W. H. Beyer (ed). 1987. CRC Handbookof Chemistry and Physics. CRC Press, Inc., Boca Raton, FL, pp. B-9.
  25. Wones, R. G., B. L. Stadler, and L. A. Frohman. 1990. Lack of effectof drinking water barium on cardiovascular risk factor. Environ.Health Perspect. 85:1-13.
  26. Zschiesche, W., K.-H. Schaller, and D. Weltle. 1992. Exposure tosoluble barium compounds: an interventional study in arc welders.Int. Arch. Occup. Environ. Health 64:13-23.

Retrieve Toxicity Profiles Condensed Version

Resources:

  1. http://chemtrails.cc/
  2. http://rense.com/general21/tox.htm)
  3. Mechanics of Geoengineering:  http://www.popularmechanics.com/science/environment/4290084




The New Humans

22 07 2013





Cosmic Consciousness Meditation by Dr. Steven Greer (5 videos)

21 07 2013

Note: “The “Im Nah Mah” phrase spoken in the begining and throughout the meditation is taken from the ancient language sanskrit used by the elder natives. It translates to “close to God” or “one with the higher being”. For full effects please watch all 5 parts in a dark room on full screen and wear sterio headphones. Hope you enjoy and Peace to my friends and subscribers.” – Youtube Channel by UFONightHawk

Part 1

Part 2

Part 3

Part 4

Part 5

Guided Meditations with Dr. Steven Greer:

{youtube=http://youtu.be/SWoGOLHMzdI]





Jupiter Sized Object Discovered Next to the Sun, Etc.

21 07 2013

by Avalon
Intellihub.com 

June 21, 2013

A spherical object the size of Jupiter was detected and imaged by the Helioviewer.org viewer on June 17 through 18. The image comes into view and is clearly spherical and looks the size of Jupiter which is approximately 86,881 miles in diameter.

For many years there’s been speculation about the existence of a dark star that is the sister sun to our own sun – making our solar system a binary–star system. According to Wikipedia; It is estimated that approximately 1/3 of the star systems in the Milky Way are binary or multiple, with the remaining 2/3 consisting of single stars.[61]

Helioviewer.org has an online viewer that can be programmed to view virtually any time you select. Images and movies can be created via the online interface. The Helioviewer Project has an online Tutorial Blog that is very useful to get started.

Some information from Wikipedia on Jupiter. our Sun and our solar system follows.

Jupiter is the fifth planet from the Sun and the largest planet in the Solar System.[11] It is a gas giant with mass one-thousandth that of the Sun but is two and a half times the mass of all the other planets in the Solar System combined. Jupiter is classified as a gas giant along with SaturnUranus and Neptune. Together, these four planets are sometimes referred to as the Jovian or outer planets

The Sun is the star at the center of the Solar System. It is almost perfectly spherical and consists of hot plasma interwoven with magnetic fields.[12][13] It has a diameter of about 1,392,684 km (865,374 mi),[5] about 109 times that of Earth, and its mass (about 2×1030 kilograms (2×1027 tons), 330,000 times that of Earth) accounts for about 99.86% of the total mass of the Solar System.[14] Chemically, about three quarters of the Sun’s mass consists of hydrogen, while the rest is mostly helium. The remainder (1.69%, which nonetheless equals 5,628 times the mass of Earth) consists of heavier elements, includingoxygencarbonneon and iron, among others.[15]

The Solar System[a] consists of the Sun and its planetary system of eight planets, theirmoons, and other non-stellar objects.[b] It formed 4.6 billion years ago from the gravitational collapse of a giant molecular cloud. The vast majority of the system’s massis in the Sun, with most of the remaining mass contained in Jupiter. The four smaller inner planets, MercuryVenusEarth and Mars, also called the terrestrial planets, are primarily composed of rock and metal. 

The Helioviewer.org online interface is where you can set parameters to view any image you select. There is also a Helioviewer.org Blog and Helioviewer.org YouTube channel.

COR1-B  (2013-06-17 00:05:18 – 2013-06-17 23:45:18 UTC)

http://helioviewer.org/?movieId=dnt95

http://helioviewer.org/api/?action=downloadMovie&id=Wnt95&format=mp4&hq=true

Source:

Helioviewer.org

H/T — BeePeeOilDisaster

Disclaimer:

The views expressed in Intellihub.com articles are the sole responsibility of the author(s) and do not necessarily reflect those of the Intellihub.com. Intellihub.com will not be held responsible or liable for any inaccurate or incorrect statements contained in Intellihub.com articles. Intellihub.com reserves the right to remove articles from the website. Posted 06–21–2013 at 13:45 PM

*****

Avalon is an Investigative Journalist and Strategist working for Intellihub.com 

The post Jupiter Sized Object Discovered Next to the Sun appeared first on Intellihub.com.

Source:  http://beforeitsnews.com/space/2013/06/jupiter-sized-object-discovered-next-to-the-sun-2461714.html





Sacred Text: Tao Te Ching

19 07 2013

This is an introduction to the Tao te Ching.  The Tao te Ching speaks to everybody on many different levels of understanding. The Tao is a book that is felt rather than intellectualized, and it can be read in its entirety in one afternoon yet contemplated for a lifetime.  Tao Te Ching Introduction – Read By Jacob Needleman

Dr. Wayne Dyer – The Wisdom of Tao Te Ching:

Dyer’s books, Manifest Your Destiny, Wisdom of the Ages, There’s a Spiritual Solution to Every Problem, and the New York Times bestsellers, 10 Secrets for Success and Inner Peace, The Power of Intention, Inspiration, Change Your Thoughts— Change Your Life, Excuses Begone, and now Wishes Fulfilled have all been featured as National Public Television specials.

Tao Te Ching by Lao Tzu (Complete)





Sheldan Nidle on Galactic Humans

19 07 2013

re:  Dimensional Shift, Hidden Human History and Human Origins, Earth’s Initiation into the Galactic Federation, Ascension, the Hollow Earth or Agartha (Ag = Inner; Artha = Earth), etc.

PAO Webinar Galactic Humans 101:

April 2013 on First Contact:

Galactic Federation Representative:

Timeline of Earth’s Hidden History According to Nidle’s Sirian Source (Metatron of Sirius):

TIMELINE

35 Million Years Ago

  • Etheric intelligent civilization created and
    are designated the guardians of Earth, but they were to find
    physical guardians also

26 Million Years Ago

  • Bipedal Dinoid with 200 IQ (from Bellatrix system in the constellation of Orion) and Bipedal Reptoid with 200 IQ colonies (from one of the lesser known stars in the constellation of Sagittarius) arrive and inhabit Earth
  • Spiritual Hierarchies of Earth send much love to these colonies
  • Mammalian species evolve to sentiency. These bipedal mammals were called the pre-cetaceans
  • Pre-cetaceans provide ample food for all 3 colonies in exchange for technology (which would in turn improve their production rate further)
  • 3 civilizations coexist in harmony trading among one another for 8 million years, all thee civilizations develop advanced forms of space/time travel
  • The pre-cetaceans develop their spiritual side extensively (psychic abilities, etc.)

10 Million Years Ago

  • Dinoid/Reptoid Alliance (from Bellatrix) ended their trade and cooperation with the pre-cetaceans – they believed they were superior and should therefore control all other beings.
  • Over the next 10,000 years the Earth Dinoids and Reptoids become more and more influenced by the Bellatrix Dinoid/Reptoid Alliance.
  • Dinoid/Reptoids decide -during these 10,000 years- to destroy the entire pre-cetacean race through various psychological war tactics
  • Pre-cetaceans sense this aggression (through their high psychic abilities) and realize the threat presented now by the Dinoid/Reptoid civilizations.

8 Million Years Ago

  • Pre-cetaceans decide to implode their fusion reactors located in the Ural Mountain range (with the Earth Spiritual Hierarchies giving them permission to do so) in a preemptive counter strike
  • Pre-cetaceans divide into two groups: one group evacuates out of our solar system (to the constellations of Pegasus and Cetus), and the other group enters the oceans to find haven. They eventually evolved over a period of 4 million years to become Earth’s present day cetaceans (i.e. the dolphins and whales)
  • Pre-cetaceans then implode their fusion reactors, thus destroying 98% of the Dinoid/Reptoid civilizations on Earth; the remaining 2% evacuated to the planet Maldek (a planet within our solar system)
  • With the Dinoid/Reptoid now no longer present on Earth, the Earth Spiritual Hierarchies and the cetaceans had to find a new suitable guardian/steward for the Earth land (since the cetaceans were guardians of Earth sea). They searched the galaxy to a distant star system 80 light years from the Sun.
  • After 2 to 3 million years of searching the galaxy, a primitive aquatic species that was starting to emerge from the oceans was discovered on the fourth planet of the Vega system. This species had creation myths, a language, and a hunting and gathering culture.
  • The Spiritual Hierarchies of the Vega system were then asked if they would permit this particular species to be vastly altered genetically to accelerate their evolution so that they may become a guardian species. The Vega Spiritual Hierarchies agreed.
  • Thus the traces of the first humans originated in the Vega star system. Their technology improved very quickly, and once they had developed star travel technology they started to migrate into nearby star systems (for a period of 2.5 million years)

4 Million Years BC

  • Galactic Federation was formed
  • Sirius B was colonized
  • Earth was selected for seeding

2 Million Years BC

  • Mars and Venus colonized
  • Hybornea colony founded on Earth

1 Million Years BC

  • Dinoid/Reptoids were building up their forces in the planet Maldek during all this time
  • Dinoids/Reptoids have a mass attack on both our solar system and also the nearby ones
  • Colonies on Earth (Hybornea), Mars and Venus were totallydestroyed.
  • Dinoids and Reptoid therefore regain control over the solar system for a period of 80,000 years.
  • In response to this the Galactic Federation planned a counterattack to reintroduce humans into this system. They arranged for a battle planet (4 times the diameter of the Earth) to come into the solar system and destroy the planet Maldek – which was the Dinoid/Reptoid stronghold. The remains of the planet Maldek are what we can now see as the asteroid belt

900,000 Years BC

  • Human colony was again founded on Earth called Lemuria (a Vedic society without rulers, laws or religion lived in Oneness with God and Nature) that lived in the 4th dimension (a higher consciousness realm) on this planet.
  • Over the next 850,000 years the Lemurians spread across the entire planet

500,000 Years BC

  • Lemurians founded what was called daughter colonies–the main ones being Atlantis, Yu (Central China and Tibet), Mu (South America), and also the Libyan/Egyptian
    colony

100,000 Years
BC

  • Atlantis, Yu and Libyan/Egyptian colonies are declared daughter empires
  • As the empires developed the Atlanteans began to acquire a feeling of uniqueness about their culture that led to feelings of separateness from the other daughter empires.
  • After some time the Atlanteans felt that THEY should become the mother empire, and quickly began to have a strong desire to destroy the Lemurians in order to gain full power.
  • The Atlanteans then began forming alliances with renegade Pleiadians and Alpha Centaurians (which had hierarchical systems of government) to further develop their technologies

25,000 Years BC

  • After waiting patiently for many years for just the right moment to attack the Lemurian empire, the Atlanteans decided to attack and destroy Lemuria with the help of their renegade Pleiadian allies.
  • They would accomplish this by taking the Earth’s other moon (Earth had 2 moons in those times) out of orbit by using force fields until it was as close as possible to the Lemurian empire, and then the moon was destroyed resulting in a catastrophic shower of meteors. This destroyed much of Lemuria, but this also resulted in many pressures being inflicted upon the tectonic plates, resulting in the gas chambers under Lemuria to implode and thus sink most of the Lemurian
    continent.

From Atlantis To The Great Flood

  • Yu empire would not bow down to the hierarchical rule of Atlantis and Libyan/Egyptian empires and was thus forced to go underground (literally). Today, they form what is known as the subterranean Kingdom of Agartha or Shamballah (not to be confused with Shambala on the Inner Spiritual Plane)
  • Power and technology-driven Atlantis forms 10 ruling districts, each with its own King. These Kings together formed the governing council of Atlantis.

25,000 – 15,000 Years BC

  • The royal governing council of Atlantis decide that a new form of government was desperately needed in which a superior ruling class could be established and sustained by their pretense that they had been empowered by a God-Force. Autocracy was thus born and was in full control enforcing a period of peace and stability.
  • The Atlanteans approved the experimenting on human genetics/DNA by master geneticists of the Pleiadian renegades to reduce the 12-strand DNA to the current double helix DNA in order to make humans more subservient and controllable (converting them into a slave race). This resulted in the dramatic reduction of consciousness, life spans, psychic/spiritual abilities, and brain usage.

15,000 – 10,000 Years BC

  • Throughout the years there were many wars among the various empires due to underground movements of people that wanted to reinstate the Lemurian “philosophy” of no hierarchy (i.e. leaders and laws). These wars led to vast destruction.
  • As a last resort the warring empires decided to attack the opponents crystal temples (which were responsible for maintaining two frozen layers of water known as Firmaments about 15,000 – 30,000 feet above ground surrounding Earth like a water base force shield which protected humans from harmful rays of the sun and also ensured a stable weather pattern at all times).
  • Unfortunately, the attacks were made simultaneously and caused the Firmament (the water layers) to be broken down and thus millions of gallons of water poured down onto the surface causing what is known biblically as “The Great Flood” (10,000 years ago)
  • The breakdown of the Firmament also resulted in the polar icecaps freezing as well as many climatic variations we have today to form.

After The Deluge

  • Only about 2 million people survived the Flood (from an original 65 million). Unfortunately many of the survivors were the mutant humans (the ones that had been genetically altered by the Pleiadians and Atlanteans into a much lower state of consciousness).
  • Also, with the destruction of the firmaments, humans were no longer protected by cosmic and atmospheric elements which resulted in further degradation of their DNA, consciousness, physique (they became smaller and shorter), brain usage and a fall of existence into the 3rd dimension.
  • A few different renegades (from Pleiades, Alpha and Beta Centauri) came to different parts of Earth after the Flood, seeing it as an opportune time to establish their own desired ideologies and also be seen as “godlike” to be revered (since there were mainly only mutant humans left, they worshipped these renegade gods). The humans were therefore easily controlled. Note that it was understood that the “heavens” from which these “gods” came was literally outer space or the cosmos.
  • These renegades established Earth’s royal houses with the concept of rulership by “divine right” (as in their heavenly or cosmic renegade ET origin–not to be confused with the Paradise Principality of The Creator). This concept of worshipping an elite has continued through to modern times whose monarchies still believe they were appointed by a divine right to rule over men. In Vedic (Lemurian) times, it was understood that no man was ruled by man.
  • Culture would rise against culture in wars claiming that the elite themselves were superior to the elite of the opposing faction–excessively ego driven.
  • We are now in times, though, that will finally bring to an end an approximate 10,000 years of sub-consciousness and regain our full consciousness that we deserve. This will be due to our entire solar system coming into contact with what is known as the the “Photon Belt

Much of his information was published almost 20 yrs ago in a book he co-wrote with Virginia Essene called “You are Becoming a Galactic Human”:

http://www.amazon.com/You-Are-Becoming-Galactic-Human/dp/0937147087








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